Profesor Tamàs Fülöp – open lectures

The Department of Physiopathology of the MUG is organising a series of open lectures to be delivered by Prof. Tamàs Fülöp from Université Sherbrooke QC Canada. The lectures are entitled Advances in gerontological research will be held from 9-12 October this year in a seminar room 23 in the building.13 (7 Dębinki Street, 1st floor next to the Doctoral Office). The initiative is aimed at students, Ph.D. students, and employees of of the MUG. Online registration available at:

– 9 October this year 11:00-13:00 – The theories of aging – link: https://szkoleniezawodowe.gumed.edu.pl/4216

– 10 October this year 11:00-13:00 – The distinction between the physiological and pathological aging: the complex approach to the biology of aging – link: https://szkoleniezawodowe.gumed.edu.pl/4217

– 11 October this year 11:00-13:00 – Immunosenescence and inflammation: how this intricate relationship can be re-conceptualised – link: https://szkoleniezawodowe.gumed.edu.pl/4218

– 12 October this year 11:13-00 – Why the Alzheimer’s disease is not caused by the amyloid beta and therefore what could cause it – link: https://szkoleniezawodowe.gumed.edu.pl/4219

Speaker’s profile

Prof. Tamàs Fülöp

Professor Tamàs Fülöp is employed as Director at the Biopharmaceutic Unit of the Research Center on Aging, University Institute of Geriatrics of Sherbrooke, University of Sherbrooke, QC Canada. He is an eminent specialist in biogerontology and immunology and is a highly experienced, practicing geriatrician at the University Institute of Geriatrics of Sherbrooke and Centre Hospitalier Universitaire de Sherbrooke. He is the author of more than 300 publications, cited more than 17600 times; his H-index=72. Prof. Fülöp is editor-in-chief of the journal Gerontology (published by Karger; IF 5.14, Q1).

The professor’s research interests lie in three main areas:

• gerontology and geriatrics (immunology of ageing, vaccination, nutritional aspects, tumourigenesis, cellular ageing);
• angiology and atherosclerosis (role of free radicals, elastase and elastin breakdown products, lipoprotein oxidation products, calcium metabolism and inflammatory processes);
• Alzheimer’s disease (immunological markers, neuroinflammation, role of pathogenic microorgnanisms, computer modelling).

The initiative is implemented under the project POWER.03.05.00-22-Z082/18.